Search results for "MESH : Mutation"

showing 9 items of 9 documents

Higher risk of death among MEN1 patients with mutations in the JunD interacting domain: a Groupe d'etude des Tumeurs Endocrines (GTE) cohort study.

2013

International audience; Multiple endocrine neoplasia syndrome type 1 (MEN1), which is secondary to mutation of the MEN1 gene, is a rare autosomal-dominant disease that predisposes mutation carriers to endocrine tumors. Although genotype-phenotype studies have so far failed to identify any statistical correlations, some families harbor recurrent tumor patterns. The function of MENIN is unclear, but has been described through the discovery of its interacting partners. Mutations in the interacting domains of MENIN functional partners have been shown to directly alter its regulation abilities. We report on a cohort of MEN1 patients from the Groupe d'étude des Tumeurs Endocrines. Patients with a…

OncologyMaleendocrine system diseasesProto-Oncogene Proteins c-jun[SDV]Life Sciences [q-bio]Diseasemedicine.disease_causeMESH: Protein Structure Tertiary0302 clinical medicineRisk FactorsMESH: Risk FactorsMESH : FemaleGenetics (clinical)MutationGeneral MedicineMESH: Follow-Up StudiesMESH : Risk Factors3. Good health030220 oncology & carcinogenesisCohortMESH : Proto-Oncogene ProteinsFemaleMESH : MutationMESH : Protein Structure TertiaryMESH : Proto-Oncogene Proteins c-junMESH : Multiple Endocrine Neoplasia Type 1Cohort studymedicine.medical_specialtyendocrine systemMESH: MutationGenetic counselingMESH : MaleMESH: Multiple Endocrine Neoplasia Type 1030209 endocrinology & metabolismBiology03 medical and health sciencesInternal medicineProto-Oncogene ProteinsGeneticsmedicineMultiple Endocrine Neoplasia Type 1HumansMEN1FamilyMolecular BiologyMESH: FamilyMESH: HumansMESH: Proto-Oncogene Proteins c-jun[ SDV ] Life Sciences [q-bio]Proportional hazards modelMESH : HumansCancerMESH : Follow-Up Studiesmedicine.diseaseMESH: MaleProtein Structure TertiaryMESH: Proto-Oncogene ProteinsMutationCancer researchMESH : FamilyMESH: FemaleFollow-Up Studies
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Diversifying selection on MHC class I in the house sparrow (Passer domesticus).

2009

10 pages; International audience; Genes of the major histocompatibility complex (MHC) are the most polymorphic loci known in vertebrates. Two main hypotheses have been put forward to explain the maintenance of MHC diversity: pathogen-mediated selection and MHC-based mate choice. Host-parasite interactions can maintain MHC diversity via frequency-dependent selection, heterozygote advantage, and diversifying selection (spatially and/or temporally heterogeneous selection). In this study, we wished to investigate the nature of selection acting on the MHC class I across spatially structured populations of house sparrows (Passer domesticus) in France. To infer the nature of the selection, we comp…

0106 biological sciencesMESH : Gene FlowMESH: Selection (Genetics)MESH: GeographyGenes MHC Class IMESH: Genetic MarkersBalancing selectionMESH : Microsatellite Repeats[ SDV.IMM.IA ] Life Sciences [q-bio]/Immunology/Adaptive immunology01 natural sciencesmicrosatellitesMESH: SparrowsMESH : Genetic MarkersMESH: AnimalsMESH: Genetic VariationMESH: Evolution MolecularGenetics0303 health scienceseducation.field_of_studyGeographybiology[SDV.BID.EVO]Life Sciences [q-bio]/Biodiversity/Populations and Evolution [q-bio.PE]MESH : GeographyMESH: Genes MHC Class I[ SDE.MCG ] Environmental Sciences/Global Changes[ SDV.BID.EVO ] Life Sciences [q-bio]/Biodiversity/Populations and Evolution [q-bio.PE][SDV.IMM.IA]Life Sciences [q-bio]/Immunology/Adaptive immunologyMate choiceMESH: Stochastic ProcessesMHC class IMESH : MutationSparrowsGene FlowGenetic MarkersMESH: Mutationbalancing selection[SDE.MCG]Environmental Sciences/Global ChangesPopulationMESH : Genetic DriftMESH: Genetics Populationchemical and pharmacologic phenomenaMESH : Stochastic ProcessesMajor histocompatibility complex010603 evolutionary biologyMESH : Genes MHC Class IEvolution Molecular03 medical and health sciencesMESH : Genetic VariationMHC class IGeneticsPasser domesticusMESH : Selection (Genetics)AnimalsMESH : Evolution MolecularSelection GeneticMESH: Genetic DrifteducationAllelesMESH: Gene FlowEcology Evolution Behavior and SystematicsSelection (genetic algorithm)030304 developmental biologyLocal adaptationIsolation by distanceStochastic Processes[ SDE.BE ] Environmental Sciences/Biodiversity and Ecologyhouse sparrowMESH: AllelesGenetic DriftGenetic Variationdiversifying selectionPasser domesticus.[ SDV.GEN.GA ] Life Sciences [q-bio]/Genetics/Animal geneticsMESH : Genetics Population[SDE.ES]Environmental Sciences/Environmental and Society[SDV.GEN.GA]Life Sciences [q-bio]/Genetics/Animal geneticsGenetics PopulationEvolutionary biologyMutationbiology.proteinMESH: Microsatellite RepeatsMESH : AnimalsMESH : Sparrows[SDE.BE]Environmental Sciences/Biodiversity and EcologyMESH : Alleles[ SDE.ES ] Environmental Sciences/Environmental and SocietyMicrosatellite Repeats
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S-nitrosylation of the death receptor fas promotes fas ligand-mediated apoptosis in cancer cells.

2011

International audience; BACKGROUND & AIMS: Fas belongs to the family of tumor necrosis factor receptors which induce apoptosis. Many cancer cells express Fas but do not undergo Fas-mediated apoptosis. Nitric oxide reverses this resistance by increasing levels of Fas at the plasma membrane. We studied the mechanisms by which NO affects Fas function. METHODS: Colon and mammary cancer cell lines were incubated with the NO donor glyceryl trinitrate or lipid A; S-nitrosylation of Fas was monitored using the biotin switch assay. Fas constructs that contained mutations at cysteine residues that prevent S-nitrosylation were used to investigate the involvement of S-nitrosylation in Fas-mediated cell…

MESH: NitroglycerinMESH: Signal TransductionTime FactorsMESH: Membrane MicrodomainsApoptosisMESH : Fas Ligand ProteinCytoplasmic partMESH: Lipid AFas ligandMiceNitroglycerin0302 clinical medicineMESH : Protein TransportMESH : FemaleMESH: AnimalsFADDLipid raft0303 health sciencesTumorbiologyColon CancerMESH : Lipid AMESH : BiotinylationGastroenterologyFas receptorMESH: Antigens CD95Protein TransportLipid AMESH : Colonic NeoplasmsMESH : Nitric OxideMESH : Nitric Oxide Donors030220 oncology & carcinogenesisColonic NeoplasmsDeath-inducing signaling complexFemale[ SDV.MHEP.HEG ] Life Sciences [q-bio]/Human health and pathology/Hépatology and GastroenterologyMESH : MutationMESH : TransfectionSignal TransductionMESH : Time FactorsMESH: Protein TransportFas Ligand ProteinMESH : Mammary Neoplasms ExperimentalMESH: MutationMESH: Cell Line TumorMESH: Mammary Neoplasms ExperimentalNitric OxideTransfectionCaspase 803 medical and health sciencesMembrane MicrodomainsCell Line TumorMESH : MiceAnimalsHumansBiotinylationNitric Oxide DonorsMESH: BiotinylationCysteinefas ReceptorMESH: MiceMESH : Protein Processing Post-Translational030304 developmental biologyMESH : Signal TransductionMESH: Colonic NeoplasmsMESH : CysteineMESH: HumansHepatologyMESH : Cell Line TumorMESH: ApoptosisMESH: TransfectionMESH : HumansMESH: Time FactorsMammary Neoplasms Experimental[SDV.MHEP.HEG]Life Sciences [q-bio]/Human health and pathology/Hépatology and GastroenterologyMESH: CysteineMESH: Nitric Oxide DonorsMolecular biologySignalingMESH: Fas Ligand ProteinMESH : NitroglycerinApoptosisLocalizationMESH: Nitric OxideMESH: Protein Processing Post-TranslationalMutationbiology.proteinMESH : Membrane MicrodomainsMESH : AnimalsMESH : Antigens CD95Protein Processing Post-TranslationalMESH: FemaleMESH : Apoptosis
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Contribution of molecular analyses in diagnosing Marfan syndrome and type I fibrillinopathies: an international study of 1009 probands.

2008

International audience; BACKGROUND: The diagnosis of Marfan syndrome (MFS) is usually initially based on clinical criteria according to the number of major and minor systems affected following international nosology. The number of FBN1 mutation carriers, at risk of aortic complications who would not be properly diagnosed based only on clinical grounds, is of growing importance owing to the increased availability of molecular screening. The aim of the study was to identify patients who should be considered for FBN1 mutation screening. METHODS: Our international series included 1009 probands with a known FBN1 mutation. Patients were classified as either fulfilling or not fulfilling "clinical"…

ProbandNosologyMarfan syndromeMalePediatricsSystemic diseaseMESH : International CooperationFibrillin-1International CooperationMESH : Aged[SDV.GEN] Life Sciences [q-bio]/GeneticsMarfan SyndromeMESH : ChildMESH: ChildEpidemiologyMESH : FemaleEctopia lentisChildGenetics (clinical)AortaAortic dissectionMESH: Aged0303 health sciences030305 genetics & heredityMicrofilament ProteinsMESH: AortaMESH : AdultConnective tissue disease3. Good healthFemaleMESH : Mutationmusculoskeletal diseasesAdultmedicine.medical_specialtycongenital hereditary and neonatal diseases and abnormalitiesMESH: MutationMESH : Microfilament ProteinsAdolescentMESH : MaleFibrillinsMESH: Marfan Syndrome03 medical and health sciencesMESH: Microfilament ProteinsMESH : AdolescentGeneticsmedicineHumans030304 developmental biologyAgedMESH: Adolescent[SDV.GEN]Life Sciences [q-bio]/GeneticsMESH : Marfan SyndromeMESH: Humansbusiness.industryMESH : HumansMESH : AortaMESH: Adultmedicine.diseaseMESH: MaleMESH: International CooperationMutation[ SDV.GEN ] Life Sciences [q-bio]/GeneticsbusinessMESH: FemaleJournal of medical genetics
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Tenectin is a novel alphaPS2betaPS integrin ligand required for wing morphogenesis and male genital looping in Drosophila.

2010

International audience; Morphogenesis of the adult structures of holometabolous insects is regulated by ecdysteroids and juvenile hormones and involves cell-cell interactions mediated in part by the cell surface integrin receptors and their extracellular matrix (ECM) ligands. These adhesion molecules and their regulation by hormones are not well characterized. We describe the gene structure of a newly described ECM molecule, tenectin, and demonstrate that it is a hormonally regulated ECM protein required for proper morphogenesis of the adult wing and male genitalia. Tenectin's function as a new ligand of the PS2 integrins is demonstrated by both genetic interactions in the fly and by cell s…

MaleMESH: Extracellular Matrix ProteinsMESH: DrosophilaMESH : Immunohistochemistry[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionIntegrinLigandsLooping morphogenesisExtracellular matrixchemistry.chemical_compound0302 clinical medicineMESH: Genitalia MaleMorphogenesisMESH: LigandsDrosophila ProteinsWings AnimalMESH: AnimalsTransgenesIn Situ Hybridization0303 health sciencesExtracellular Matrix ProteinsMESH : Genitalia MaleMESH : LigandsIntegrin alpha ChainsCell adhesion moleculeMESH : In Situ HybridizationImmunohistochemistry3. Good healthCell biologyLarvaMESH : Integrin alpha ChainsAdhesionDrosophilaMESH : MutationMESH : TransgenesTenectinIntegrin alpha ChainsDrosophila ProteinEcdysoneEcdysoneMESH: MutationMESH: Drosophila ProteinsMESH : MaleIntegrinMorphogenesisMESH : WingMESH: TransgenesBiologyGenitalia MaleArticle03 medical and health sciencesMESH : Extracellular Matrix ProteinsMESH: In Situ HybridizationAnimalsMESH : DrosophilaCell adhesionMolecular Biology030304 developmental biologyMESH : LarvaMetamorphosisMESH: Integrin alpha ChainsLeft–right asymmetryMESH: ImmunohistochemistryCell BiologyMESH : Drosophila ProteinsMESH: WingMESH: MaleMESH: MorphogenesischemistryMESH : MorphogenesisMutationbiology.proteinMESH : AnimalsMESH: Larva[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgeryDevelopmental Biology
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The consequences of regulation of desat1 expression for pheromone emission and detection in Drosophila melanogaster.

2010

AbstractSensory communication depends on the precise matching between the emission and the perception of sex- and species-specific signals; understanding both the coevolutionary process and the genes involved in both production and detection is a major challenge. desat1 determines both aspects of communication—a mutation in desat1 simultaneously alters both sex pheromone emission and perception in Drosophila melanogaster flies. We investigated whether the alteration of pheromonal perception is a consequence of the altered production of pheromones or if the two phenotypes are independently controlled by the same locus. Using several genetic tools, we were able to separately manipulate the tw…

Fatty Acid DesaturasesMaleTranscription Genetic[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionMESH : Animals Genetically ModifiedMESH : GenotypeMESH: GenotypeAnimals Genetically ModifiedSexual Behavior AnimalMESH : HydrocarbonsMESH: Reverse Transcriptase Polymerase Chain ReactionMESH : Drosophila melanogasterDrosophila ProteinsMESH: AnimalsMESH : FemaleMESH: Sexual Behavior AnimalSex AttractantsGeneticsMESH: Nursing AssessmentMESH : Craniocerebral TraumabiologyMESH : Gene Expression RegulationReverse Transcriptase Polymerase Chain ReactionMESH : Fatty Acid DesaturasesMESH : Reverse Transcriptase Polymerase Chain ReactionMESH: Fatty Acid DesaturasesMESH: Gene Expression RegulationPhenotypeMESH: Intracranial PressureMESH: Sex AttractantsDrosophila melanogasterSex pheromonePheromoneFemaleDrosophila melanogasterMESH : MutationMESH: MutationGenotypeMESH : ComaMESH: Drosophila ProteinsMESH : MaleMESH: Craniocerebral TraumaSensory systemLocus (genetics)InvestigationsMESH: Drosophila melanogasterMESH: Animals Genetically ModifiedMESH: HydrocarbonsMESH: Education Nursing ContinuingGeneticsMESH : Nursing AssessmentAnimalsMESH : Sexual Behavior AnimalGeneMESH: ComaTranscriptional activityMESH : Sex AttractantsMESH: HumansMESH: Transcription GeneticMESH : HumansMESH : Transcription Geneticbiology.organism_classificationMESH : Drosophila ProteinsMESH: MaleHydrocarbonsMESH : Intracranial PressureGene Expression RegulationMutationMESH : AnimalsMESH : Education Nursing ContinuingMESH: Female[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition
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Genes involved in sex pheromone discrimination in Drosophila melanogaster and their background-dependent effect.

2012

International audience; Mate choice is based on the comparison of the sensory quality of potential mating partners, and sex pheromones play an important role in this process. In Drosophila melanogaster, contact pheromones differ between male and female in their content and in their effects on male courtship, both inhibitory and stimulatory. To investigate the genetic basis of sex pheromone discrimination, we experimentally selected males showing either a higher or lower ability to discriminate sex pheromones over 20 generations. This experimental selection was carried out in parallel on two different genetic backgrounds: wild-type and desat1 mutant, in which parental males showed high and l…

MaleMESH: Olfactory Perception[ SDV.AEN ] Life Sciences [q-bio]/Food and NutritionMESH : Animals Genetically Modifiedlcsh:MedicineGenes InsectMESH: Genes InsectBreedingMESH : Behavior AnimalMESH: ReproductionCourtshipAnimals Genetically ModifiedSexual Behavior Animal0302 clinical medicineMESH : Drosophila melanogasterMESH: Behavior AnimalMESH : FemaleMESH: AnimalsMatingSex AttractantsMESH: Sexual Behavior Animal10. No inequalitylcsh:Sciencemedia_commonGenetics0303 health sciencesMultidisciplinaryEcologyBehavior AnimalReproductionMESH : Genes InsectAnimal ModelsMESH : ReproductionSensory SystemsDrosophila melanogasterMESH: Sex AttractantsMate choiceSex pheromoneAlimentation et NutritionFemaleDrosophila melanogasterMESH : MutationResearch ArticleMESH: Mutationmedia_common.quotation_subjectMESH : BreedingMESH : MaleMESH: CourtshipContext (language use)MESH: BreedingBiologyMESH: Drosophila melanogasterMESH: Animals Genetically Modified03 medical and health sciencesModel OrganismsSpecies SpecificityMESH : Olfactory PerceptionGeneticsFood and NutritionAnimalsMESH : Species SpecificityMESH: Species SpecificityAlleleMESH : Sexual Behavior AnimalBiology030304 developmental biologyEvolutionary BiologyMESH : Sex AttractantsAnimals;Animals;Genetically Modified;Behavior;Animal;Breeding;Courtship;Drosophila melanogaster;Female;Genes;Insect;Male;Mutation;Olfactory Perception;Reproduction;Sex Attractants;Sexual Behavior;Species SpecificityMESH : Courtshiplcsh:RCourtshipbiology.organism_classificationOlfactory PerceptionMESH: MaleMutationSex Attractantslcsh:QMESH : AnimalsMESH: Female[SDV.AEN]Life Sciences [q-bio]/Food and Nutrition030217 neurology & neurosurgeryNeurosciencePLoS ONE
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Arabidopsis thaliana nicotianamine synthase 4 is required for proper response to iron deficiency and to cadmium exposure.

2013

International audience; The nicotianamine synthase (NAS) enzymes catalyze the formation of nicotianamine (NA), a non-proteinogenic amino acid involved in iron homeostasis. We undertook the functional characterization of AtNAS4, the fourth member of the Arabidopsis thaliana NAS gene family. A mutant carrying a T-DNA insertion in AtNAS4 (atnas4), as well as lines overexpressing AtNAS4 both in the atnas4 and the wild-type genetic backgrounds, were used to decipher the role of AtNAS4 in NA synthesis, iron homeostasis and the plant response to iron deficiency or cadmium supply. We showed that AtNAS4 is an important source for NA. Whereas atnas4 had normal growth in iron-sufficient medium, it dis…

0106 biological sciences[ SDV.BV ] Life Sciences [q-bio]/Vegetal BiologyMESH : Azetidinecarboxylic AcidFMN ReductaseArabidopsis thalianaMutantArabidopsisGene ExpressionPlant Science01 natural sciencesMESH : Cation Transport ProteinsMESH : IronMESH : Arabidopsis ProteinsNicotianamine synthaseMESH : Plants Genetically Modifiedchemistry.chemical_compoundMESH : ArabidopsisGene Expression Regulation PlantGene expressionMESH: Genes PlantArabidopsis thalianaMESH : DNA BacterialHomeostasisMESH: ArabidopsisNicotianamineMESH: Stress PhysiologicalCation Transport ProteinsMESH : Adaptation PhysiologicalMESH : Cadmium2. Zero hungerchemistry.chemical_classification0303 health sciencesCadmiumMESH: IronbiologyGeneral MedicineIron DeficienciesPlants Genetically ModifiedAdaptation PhysiologicalMESH: Azetidinecarboxylic AcidMESH : PhenotypePhenotypeBiochemistryMESH: HomeostasisMESH : HomeostasisMESH : MutationAzetidinecarboxylic AcidCadmiumDNA BacterialMESH: Gene ExpressionMESH: MutationIronMESH: Cadmiumchemistry.chemical_elementMESH: FerritinsMESH: Arabidopsis ProteinsMESH: Alkyl and Aryl TransferasesGenes PlantMESH: PhenotypeNicotianamine synthase03 medical and health sciencesMESH: Cation Transport ProteinsStress PhysiologicalIron homeostasisGenetics[SDV.BV]Life Sciences [q-bio]/Vegetal BiologyIron deficiency (plant disorder)MESH: Gene Expression Regulation PlantMESH : Genes PlantMESH : Alkyl and Aryl TransferasesMESH : Stress Physiological030304 developmental biologyMESH : FMN ReductaseAlkyl and Aryl TransferasesArabidopsis ProteinsIron deficiencyNitric oxideNicotianaminebiology.organism_classificationMESH: Adaptation PhysiologicalMESH: DNA BacterialMESH : Gene ExpressionEnzymechemistryMESH: FMN ReductaseMESH: Plants Genetically ModifiedFerritinsMutationbiology.proteinMESH : FerritinsAgronomy and Crop ScienceMESH : Gene Expression Regulation Plant010606 plant biology & botany
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The FBN2 gene: new mutations, locus-specific database (Universal Mutation Database FBN2), and genotype-phenotype correlations.

2009

International audience; Congenital contractural arachnodactyly (CCA) is an extremely rare disease, due to mutations in the FBN2 gene encoding fibrillin-2. Another member of the fibrillin family, the FBN1 gene, is involved in a broad phenotypic continuum of connective-tissue disorders including Marfan syndrome. Identifying not only what is in common but also what differentiates these two proteins should enable us to better comprehend their respective functions and better understand the multitude of diseases in which these two genes are involved. In 1995 we created a locus-specific database (LSDB) for FBN1 mutations with the Universal Mutation Database (UMD) tool. To facilitate comparison of …

Fibrillin-2MESH : Polymorphism GeneticFibrillin-1DNA Mutational AnalysisMESH : Genotype[SDV.GEN] Life Sciences [q-bio]/Geneticscomputer.software_genreMESH: Genotype0302 clinical medicineGenotypeDatabases GeneticMissense mutationCongenital contractural arachnodactylyMESH: DNA Mutational AnalysisGenetics (clinical)MESH: Databases GeneticRegulation of gene expressionGenetics0303 health sciencesDatabaseMESH : Gene Expression RegulationMicrofilament ProteinsPhenotypeMESH: Gene Expression RegulationBeals-Hecht syndrome3. Good healthINCMESH : PhenotypePhenotypeMESH : MutationFibrillinmusculoskeletal diseasesMESH: MutationGenotypeMESH : Microfilament Proteinsdatabase OFFICIAL JOURNAL wwwhgvsorg & 2008 WILEY-LISSLocus (genetics)fibrillinMESH : DNA Mutational AnalysisBiologyFibrillinsMESH: PhenotypeMESH: Sequence Homology Nucleic Acidcongenital contractural arachnodactyly03 medical and health sciencesMESH: Microfilament ProteinsSequence Homology Nucleic AcidMESH: Polymorphism GeneticGeneticsmedicineHumansMESH : Sequence Homology Nucleic AcidFBN2CCAMESH : Databases GeneticGene030304 developmental biology[SDV.GEN]Life Sciences [q-bio]/GeneticsPolymorphism GeneticMESH: HumansMESH : Humansmedicine.diseaseGene Expression RegulationMutation[ SDV.GEN ] Life Sciences [q-bio]/Geneticscomputer030217 neurology & neurosurgery
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